, 2001). This constrained malignant development relates prominently towards the activation of p53 in aspiring cancer cells encountering telomere dysfunction. Inactivation on the p53 tumor suppressor is The most frequent functions in human epithelial cancers (Brosh and Rotter, 2009) along with the status of p53 can dictate whether or not https://klausl875uhr6.win-blog.com/profile